Luiz Tarcisio B. Filomeno I ; Clara R. Carelli II ; Nuno C. Descritores: Embolia gordurosa; Artroplastia; Fraturas.
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Paroxysmal sympathetic hyperactivity represents an uncommon and potentially life-threatening complication of severe brain injuries, which are most commonly traumatic. This syndrome is a clinical diagnosis based on the recurrent occurrence of tachycardia, hypertension, diaphoresis, tachypnea, and occasionally high fever and dystonic postures.
The episodes may be induced by stimulation or may occur spontaneously. Underdiagnosis is common, and delayed recognition may increase morbidity and long-term disability. Trigger avoidance and pharmacological therapy can be very successful in controlling this complication. Fat embolism syndrome is a rare but serious complication of long bone fractures.
Neurologic signs, petechial hemorrhages and acute respiratory failure constitute the characteristic presenting triad. The term cerebral fat embolism is used when the neurological involvement predominates. The diagnosis is clinical, but specific neuroimaging findings can be supportive. The neurologic manifestations include different degrees of alteration of consciousness, focal deficits or seizures. Management is supportive, but good outcomes are possible even in cases with very severe presentation.
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Which day? Send at most: 1 item 5 items 10 items 20 items 50 items items items. Send even when there aren't any new results. Optional text in email:. Save Cancel. Create a file for external citation management software Create file Cancel. Full-text links Cite Favorites. Abstract in English , Portuguese. Conflict of interest statement Conflicts of interest: None. Figures Figure 1 5 Images of patient 1 at….
Figure 1 13 Images of patient 1 at admission to the emergency room. A Multi-fragmentary fractures…. Images of patient 1 at admission to the emergency room. A Multi-fragmentary fractures of the lower third of right tibia and fibula.
B Computed tomography scan showing bilateral frontal subcortical hypodense areas without a midline shift. No hemorrhage was evident, and the basal cisterns and sulcus remained visible. C Magnetic resonance image showing multiple lesions in both cerebral hemispheres that were hypointense on T1 and hyperintense on T2 and FLAIR sequences in the periventricular white matter of both frontoparietal regions.
Figure 2 5 Patient 2 images at admission. Figure 2 13 Patient 2 images at admission. A Closed fracture of the lower third of…. Patient 2 images at admission. A Closed fracture of the lower third of the tibia. B Computed tomography scan showing subcortical hypodense lesions in the frontal and left parietal regions. C-D Magnetic resonance images showing multiple T2-hyperintense lesions localized in periventricular white matter and bilateral frontal and parietal subcortical regions.
See this image and copyright information in PMC. Similar articles Early diagnosis of paroxysmal sympathetic hyperactivity in the ICU. Hughes JD, et al. Neurocrit Care. PMID: Susceptibility weighted imaging in a patient with paroxysmal sympathetic storms.
Huang P, et al. J Neurol. Epub Mar 6. PMID: No abstract available. Paroxysmal sympathetic hyperactivity after severe brain injury. Lump D, Moyer M. Lump D, et al. Curr Neurol Neurosci Rep. PMID: Review. Alternating hemidystonia following traumatic brain injury as an unusual presentation of paroxysmal autonomic instability with dystonia syndrome. Buerger KJ, Salazar R. Buerger KJ, et al. BMJ Case Rep.
Paroxysmal autonomic instability with dystonia after brain injury. Blackman JA, et al. Arch Neurol. Show more similar articles See all similar articles. Morales-Vidal SG. References Lump D, Moyer M. A review of paroxysmal sympathetic hyperactivity after acquired brain injury. Ann Neurol.
Paroxysmal sympathetic hyperactivity in severe traumatic brain injury. Acta Neurochir Wien ; 11 — Understanding paroxysmal sympathetic hyperactivity after traumatic brain injury. Surg Neurol Int. A critical review of the pathophysiology of dysautonomia following traumatic brain injury.
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Paroxysmal Sympathetic Hyperactivity Syndrome Caused by Fat Embolism Syndrome
Paroxysmal sympathetic hyperactivity represents an uncommon and potentially life-threatening complication of severe brain injuries, which are most commonly traumatic. This syndrome is a clinical diagnosis based on the recurrent occurrence of tachycardia, hypertension, diaphoresis, tachypnea, and occasionally high fever and dystonic postures. The episodes may be induced by stimulation or may occur spontaneously. Underdiagnosis is common, and delayed recognition may increase morbidity and long-term disability.
Embolia gordurosa encefálica
Skip to search form Skip to main content You are currently offline. Some features of the site may not work correctly. DOI: A embolia gordurosa e constituida pela presenca de globulos de gordura dentro da microcirculacao periferica e pulmonar, com ou sem quadro clinico. View PDF. Save to Library. Create Alert.
Fat Embolism Syndrome in Femoral Shaft Fractures: Does the Initial Treatment Make a Difference?
Fat embolism : a review for current orthopaedics practice. Acta ortop. ISSN Fat embolism FE is the occlusion of small blood vessels by fat droplets originated mainly from femur, tibia and pelvis fractures, as well as from knee and hip arthroplasty. It usually does not cause damage to the involved organs, unless when it is massive. In a few cases, FE evolves to the 'fat embolism syndrome' FES , affecting most often the lungs and the brain, although any organ or structure of the body can be damaged.